Procyanidin A1 and its digestive products inhibit acrylamide-induced IPEC-J2 cell damage: apoptosis and cell cycle arrest

Author(s)
Fangfang Yan, Si Quin, Chengmin Wang, Wolfram Weckwerth, Qun Lu, Rui Liu
Abstract

Our previous study has demonstrated that procyanidin A1 (A1) and its simulated digestive product (D-A1) can prevent acrylamide (ACR)-induced cytotoxicity in small intestine cells. However, the potential mechanism remains poorly understood. In this study, ACR treatment was found to increase the levels of 8-hydroxy-deoxyguanine (8-OHdG) and phosphorated histone H2AX (γH2AX), two DNA damage markers, thereby resulting in cell cycle arrest in the G2/M phase; whereas both A1 and D-A1 could prevent the phosphorylation of ataxia telangiectasia mutated (ATM) and checkpoint kinase 2 (Chk2), and then regulate the expression of G2/M phase-related proteins, finally maintaining normal cell cycle progression. Moreover, A1 and D-A1 could increase the B cell lymphoma 2 (Bcl-2)/Bcl2-associated X (Bax) ratio and decrease the expression of cleaved caspase-3 and cleaved caspase-9 proteins to alleviate ACR-induced cell apoptosis, which might be related to the inhibition of the mitogen-activated protein kinase (MAPK) pathway. More importantly, A1 showed no remarkable variation in inhibitory effect before and after digestion, indicating that it can endure gastrointestinal digestion and may be a promising phytochemical to alleviate ACR-induced intestinal cell damage.

Organisation(s)
Functional and Evolutionary Ecology
External organisation(s)
Hunan Agricultural University, Huazhong Agricultural University
Journal
Food Science and Human Wellness
ISSN
2213-4530
DOI
https://doi.org/10.26599/FSHW.2024.9250272
Publication date
09-2024
Peer reviewed
Yes
Austrian Fields of Science 2012
301303 Medical biochemistry
Keywords
ASJC Scopus subject areas
Biochemistry, Genetics and Molecular Biology(all)
Sustainable Development Goals
SDG 3 - Good Health and Well-being
Portal url
https://ucrisportal.univie.ac.at/en/publications/3f2a74ac-6fe3-4745-93c9-9c4ea4f0be91